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Response to: Correspondence on ‘Role of AMPK/mTOR-independent autophagy in clear cell renal cell carcinoma’ by Lorzadeh et al
  1. Milan Radovanovic1,
  2. Sasenka Vidicevic2,
  3. Jelena Tasic2,
  4. Nina Tomonjic2,
  5. Zeljka Stanojevic2,
  6. Predrag Nikic1,
  7. Aleksandar Vuksanovic1,
  8. Zoran Dzamic1,
  9. Uros Bumbasirevic1,
  10. Aleksandra Isakovic2,
  11. Vladimir Trajkovic3
  1. 1 Urology Clinic, Clinical Center of Serbia, Belgrade, Serbia
  2. 2 Institute of Medical and Clinical Biochemistry, Faculty of Medicine, University of Belgrade, Belgrade, Serbia
  3. 3 Institute of Microbiology and Immunology, Faculty of Medicine, University of Belgrade, Belgrade, Serbia
  1. Correspondence to Professor Vladimir Trajkovic, Institute of Microbiology and Immunology, University of Belgrade Faculty of Medicine, Belgrade, Serbia; vladimir.trajkovic{at}med.bg.ac.rs

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Dear Editor,

We read with great interest the commentary by Lorzadeh and Ghavami1 on our article that reported AMP-activated protein kinase (AMPK)/mechanistic target of rapamycin (mTOR)-independent upregulation of autophagy in the tumor tissue of clear cell renal cell carcinoma (ccRCC) patients.2 We agree with their observation that the somewhat surprising absence of a link between AMPK/mTOR signaling and autophagy induction in ccRCC …

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Footnotes

  • Contributors VT wrote the manuscript; MR, SV, JT, NT, ZS, PN, AV, ZD, UB, and AI provided critical feedback and contributed to the final manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Provenance and peer review Commissioned; internally peer reviewed.

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